Jeremy A. MacMahon, Phd
Scientist
Jeremy MacMahon joined Intertox in August 2024 as a staff scientist. His work has primarily focused on providing industry and litigation support related to dermal exposures to chemicals in textiles and airborne environmental exposure to mold and bacteria.
Expertise:
Dr. MacMahon's expertise includes:
- Conducting risk assessments for airborne and surface exposures to various mold and bacteria.
- Pharmacokinetic modeling of ethanol metabolism and intoxication.
- Creating complex large-scale databases to analyze, model, and present data effectively.
- Communicating complex scientific ideas to diverse populations.
As a science communicator, he has published a number of non-refereed articles tackling difficult scientific ideas across toxicology with topics including COVID-19, environmental exposures from natural gas, military burn pits, and shellfish consumption, and tattoo toxicity. These articles focused on communicating comprehensive analysis of events with serious toxicological implications to groups of people without a scientific background.
In addition to this work, Mr. MacMahon has conducted research on anti-epileptogenic and disease modifying drugs used to treat chemically induced epilepsy. These studies included leveraging animal models to conduct behavioral, biochemical, in vivo imaging, and immunohistochemical assays using cutting edge techniques and analysis. An image from these studies was featured on the cover of the journal NeuroToxicology, volume 104.
Email: jmacmahon@intertox.com
Education:
Dr. MacMahon earned his Ph.D. in 2024 from University of California: Davis, School of Veterinary Medicine, and earned his Bachelor of Science degree in Biochemistry and Molecular Biology from University of California: Davis in 2019. His dissertation for the Pharmacology and Toxicology graduate program explored the therapeutic potential of soluble epoxide hydrolase inhibition to treat long term behavioral, pathological, and cognitive symptoms of epilepsy in a rat model of pesticide induced status epilepticus.